Transurethral Resection of Bladder Tumour (TURBT) Surgery Waiting Times Update
Find out about the latest waiting times and length of hospital stay for transurethral resection of bladder tumour (TURBT) surgery with Dr Foster Health
Transurethral Resection of Bladder Tumour (TURBT) summary
Transurethral Resection of Bladder Tumour (TURBT) is an operation to remove superficial (non-invasive) bladder cancer. The following section clarifies this condition and resulting medical procedures.
What is the bladder?
The urinary bladder is basically a hollow, muscular bag. Urine drains into the bladder from the kidneys through two tubes (ureters) and can be stored there or expelled by muscular contractions through a tube (urethra) to the outside of the body.
Find out more about Bladder Cancer by visiting Dr Foster Health's Medical Dictionary
What is bladder cancer?
Bladder cancer occurs when a tumour grows in the lining of the bladder. A tumour develops when cells divide abnormally and bunch together to form a lump, which, if malignant (cancerous), can spread to other parts of the body.
The two main types of bladder cancer:
- Non-invasive - the cancer is contained within cells that form the inner lining of the bladder
- Invasive - the cancer has spread to the muscle layer of the bladder or to other organs
TURBT operation for non-invasive bladder cancer
During a TURBT operation, the surgeon uses a surgical tool called a resectoscope - a long thin tube with a tiny camera and light source at one end - to remove cancerous tumour(s) from the bladder.
The surgeon will pass the thin tube of the resectoscope down the urethra and into the bladder. The surgeon will then be able to see what is happening inside the bladder by watching a monitor where the images from the camera are displayed.
Surgical instruments inside the resectoscope access the bladder and the abnormal area is then resected (removed).
Waiting times for TURBT
Government cancer targets have gone some way to reducing waiting times for TURBT.
Dr Foster Health data shows that waiting times for TURBT have generally been falling year-on-year in all Strategic Health Authorities (SHA) since 2005/6:
Fig 3 - Average waiting times for TURBT by Strategic Health Authority (SHA)
Across all 10 Strategic Health Authorities, the average waiting time for a TURBT operation in 2008/9 is about 33 days. You will wait the shortest time for a TURBT (28 days) if you live in the South Central SHA. You will have the longest wait (37 days) if you live in the London SHA.
What is a Strategic Health Authority?
Strategic Health Authorities (SHAs) were formed by the government in 2002 to manage the local NHS on behalf of the Secretary of State. There are now 10 SHAs whose key role is to act as a link between the Department of Health and the NHS.
Hospital length of stay for TURBT
The following graph shows you the average length of stay in hospital after TURBT surgery according to age group.
Fig 4 - TURBT: Spells & length of stay by Age Group

Inpatient spell: a patient's stay in hospital consists of at least one finished consultant episode (period of care under one consultant/team)
Figure 4 shows that as patients get older they are more likely to stay in hospital for longer after a TURBT procedure.
Across all age groups, the average length of stay in 2008/9 is between two and three days in hospital.
New developments in the treatment of bladder cancer
The following medical treatments are under investigation in clinical trials and may not be available at NHS or private hospitals unless you are part of a clinical trial. These treatments may become available at a later date, if researchers can demonstrate they are clinically safe and effective.
- Genetic changes in bladder cancer - a number of clinical studies are looking at different tests that can detect DNA abnormalities and help doctors predict the prognosis (how successful the treatment will be) of bladder cancer patients as well as choose the appropriate treatment for a particular patient (1)
- Prevention of bladder cancer - even if your bladder cancer was treated successfully, you are at risk of developing a new cancer in the bladder, lining of the kidneys, ureters and urethra. Studies are looking at the properties of specific vitamins or minerals which may reduce the risk of a second cancer (2). Further studies are looking at a non-steroidal anti-inflammatory drug called celecoxib, while other researchers are looking for a vaccine to help lower the risk of developing a second cancer (3)
- Photodynamic therapy (PDT) - PDT is a potentially useful treatment for non-invasive bladder cancer that kills cancer cells in a targeted way (4). The patient is injected with a harmless chemical, which builds up in the bladder over several days. Then a special type of laser light is focused on the bladder through a cystoscope. The laser light changes the chemical that has collected in the cancer cells into a new chemical that can kill them
- Chemotherapy - a number of chemotherapy drugs are being tested as treatment for bladder cancer. Some of these, such as irinotecan (5) and oxaliplatin (6) are already used to treat other types of cancer
- Anti-angiogenesis drugs - anti-angiogenesis drugs slow down cancers by starving the tumour/s of blood. Bevacizumab (7) and sorafenib (8) have been effective against colorectal cancer and kidney cancer, and are currently being tested in patients with bladder cancer
- Targeted therapies - a number of studies are investigating the molecules within bladder cancer cells which control their growth and spread to develop new targeted treatments. Some of the drugs are used to treat other cancers, such as lung and colon cancer (9). A number of clinical trials are testing the following drugs to fight bladder cancer: lapatinib (10); erlotinib (11); trastuzumab (12); and gefitinib (13)
- Gene therapy - Gene therapy uses special viruses that have been modified in the laboratory. The modified viruses are injected into the bladder and inject selected genes into the cells of the bladder. For example, the GM-CSF gene helps the cells produce an immune system hormone (cytokine) that researchers hope will activate the immune system to attack the cancer (14/15)
References:
- Bellmunt, Paz-Ares & Cuello et al | Gene expression of ERCC1 as a novel prognostic marker in advanced bladder cancer patients receiving cisplatin-based chemotherapy | Annals of Oncology | DOI:10.1093/annonc/mdl435
- Rayman | Selenium in cancer prevention: a review of the evidence and mechanism of action | Proceedings of the Nutrition Society (2005), 64 : 527-542 | DOI:10.1079/PNS2005467 | Published online by Cambridge University Press (Mar 2007)
- Dhawan, Borgatti & Jeffreys et al | Cyclooxygenase-2 dependent and independent antitumor effects induced by celecoxib in urinary bladder cancer cells | Molecular Cancer Therapeutics (Apr 2008): (7), p 897 | DOI: 10.1158/1535-7163.MCT-07-0313
- Mejia & Nseyo | Safety of three sequential whole bladder photodynamic therapy (WBPDT) treatments in the management of resistant bladder cancer (Proceedings Paper) | Photonic Therapeutics and Diagnostics (Feb 2009. Proceedings Vol. 7161)
- Beer, Goldman & Nichols et al | Phase II Study of Irinotecan in Patients with Advanced Transitional Cell Carcinoma of the Urothelium that Progressed After Platinum-Based Chemotherapy | Journal Clinical Genitourinary Cancer (Mar 2008): (6); 1; pp 36-39 | ISSN: 1558-7673 (Print)/ 1938-0682 (Online) | DOI: 10.3816/CGC.2008.n.006
- Mir, Alexandre & Ropert et al | Oxaliplatin in inoperable or metastatic bladder cancer | Annals of Oncology 2006 17 (12): pp 1853-1854 | DOI:10.1093/annonc/mdl157
- Osai, Ng & Pagliaro et al | Positive response to bevacizumab in a patient with metastatic, chemotherapy-refractory urothelial carcinoma | Anti-Cancer Drugs (Apr 2008): (19); Issue 4; pp 427-429 | DOI: 10.1097/CAD.0b013e3282f52bef
- Gollob, Rathmell & Richmond et al | Phase II Trial of Sorafenib Plus Interferon Alfa-2b As First- or Second-Line Therapy in Patients With Metastatic Renal Cell Cancer | Journal of Clinical Oncology (Aug 2007): (25); No 22; pp 3288-3295 | DOI: 10.1200/JCO.2007.10.8613
- Black, Agarwal & Dinney | Targeted therapies in bladder cancer-an update | Journal of Urologic Oncology (Sep 2007): (25); 5; pp 433-438 | PII: S1078-1439(07)00144-5 | DOI: 10.1016/j.urolonc.2007.05.011
- McHugh, Kriajevska & Mellon et al | Combined Treatment of Bladder Cancer Cell Lines with Lapatinib and Varying Chemotherapy Regimens-Evidence of Schedule-Dependent Synergy | Journal of Urology (Feb 2007): (69); 2; pp 390-394 | PII S0090-4295(06)02627-6
- Smith, Coward & Kim | A phase II study of neoadjuvant erlotinib (Tarceva) in patients with muscle-invasive bladder cancer undergoing radical cystectomy: Preliminary results | Journal of the American College of Surgeons (Sep 2009): (209), 3, (Supplement), p S129 | PII: S1072-7515(09)00900-4 | DOI: 10.1016/j.jamcollsurg.2009.06.323
- Beuzeboc, Banu & Voog | Trastuzumab combined with standard chemotherapy in HER+ metastatic bladder cancer patients: Interim safety results of a prospective randomized phase II study | Journal of Clinical Oncology (Jun 2007 Supplement): ASCO Annual Meeting Proceedings (Post-Meeting Edition): Vol 25, No 18S 2007: 15565
- Kassouf, Brown & Black et al | Is Vascular Endothelial Growth Factor Modulation a Predictor of the Therapeutic Efficacy of Gefitinib for Bladder Cancer? | The Journal of Urology (Sep 2008): (180), 3, pp 1146-1153 | PII: S0022-5347(08)01221-4 | DOI: 10.1016/j.juro.2008.05.001
- Rein, Breidenbach & Curiel | Current developments in adenovirus-based cancer gene therapy | Journal of Future Oncology (Feb 2006): (2); 1; pp 137-143 | DOI 10.2217/14796694.2.1.137
- Cross & Burmester | Gene Therapy for Cancer Treatment: Past, Present and Future | Journal of Clinical Medicine & Research (Sep 2006); (4); 3: pp 218 -227 | DOI:10.3121/cmr.4.3.218
